If you are concerned about your cholesterol, you need to watch this two part video on Australian TV by Maryanne Demasi (upper left image).. Each segment is 30 minutes.
Our routine lab sheet includes a cholesterol panel which we review with every patient.
“I am worried about my cholesterol. What is it ? Is my cholesterol too high?” ,
“My cholesterol is higher than the lab range? Should I be worried?”
“My other doctor says my cholesterol is too high and I need a statin drug. What should I do?.
“How can I lower my cholesterol ?”
Above left image courtesy of Maryanne Demasi.Australian Journalist and Celebrity who produced a new video on Cholesterol..
Some people have a low serum cholesterol, yet are not protected from heart disease. In spite of the low cholesterol report they go on to repeated heart attacks. Others have high cholesterol, yet seem to be fine with totally clean arteries, and no heart disease. Finally there are the genetic familial hypercholesterolmeia patients. Some of these people live into their 60’s in perfect health, without the slightest hint of heart disease, yet have very high cholesterol above 300 their entire lives..
These are the people who have very high cholesterol levels, in the 300-350 range their entire lives, from a genetic mutation in the LDL receptor.
The Simon Broome Familial Hypercholesterolemia Registry
The Simon Broome Familial Hypercholesterolemia Registry reports their experience treating Familial Hypercholesterolemia with statin drugs to lower cholesterol. Yet, hidden in the report is a nugget of information about a sub-group over the age of 60 with high cholesterol (over 300-350) their entire lives. They go on to say that their data shows these people are a “highly selected group with no increased risk of coronary artery disease“ !!! (3,4) This is astounding ! They report life long high cholesterol of 350, yet no heart disease in this group.!! The existence of this group proves that the Cholesterol Theory is a Myth, and cholesterol is NOT the culprit which causes heart disease.
Maryanne Demasi, an Australian journalist and TV Producer, has compiled a two part video with interviews of cardiologists, lipidologists and other experts on the question of cholesterol and heart disease. Part one discusses the Cholesterol Theory .and Part Two the Statin anti-cholesterol drugs. She interviews Rita Redberg MD, Beatrice Golomb MD, Jon Abramson MD and Stephen SInatra MD in part two.
Thanks to Uffe Ravnskov MD PhD of Thincs for bringing this to my attention.
Articles with Related Interest
Heart of the Matter. Does high cholesterol really increase your risk of heart attacks? Thursday 24 October 8pm on ABC 1
Is the role of cholesterol in heart disease really one of the biggest myths in the history of medicine? For the last four decades we’ve been told that saturated fat clogs our arteries and high cholesterol causes heart disease. It has spawned a multi-billion dollar drug and food industry of “cholesterol free” products promising to lower our cholesterol and decrease our risk of heart disease. But what if it all isn’t true? What if it’s never been proven that saturated fat causes heart disease? In a special two part edition of Catalyst, Dr Maryanne Demasi investigates the science behind the claims that saturated fat causes heart disease by raising cholesterol.
Simon Broome Familial Hyperlipidaemia Register Group 2008
In the heterozygous condition, the cumulative risk of a coronary event by the age of 60 years without effective treatment is at least 50% in men and ∼30% in women. Coronary disease occurs ∼10 years earlier in men than in women, with a marked increase in women post-menopausally.4–6
Coronary mortality: We found that, before and after statins became widely available, there was no excess coronary mortality in patients aged >60 years without known coronary disease at registrationPatients surviving into older age before statins became available were therefore likely to be a highly selected group at lower risk of coronary disease.
BMJ. 1991 October 12; 303(6807): 893–896.
Risk of fatal coronary heart disease in familial hypercholesterolaemia. Scientific Steering Committee on behalf of the Simon Broome Register Group.
OBJECTIVES–(a) To determine the excess mortality from all causes and from coronary heart disease in patients with familial hypercholesterolaemia; (b) to examine how useful various criteria for selective measurement of cholesterol concentration in cardiovascular screening programmes are in identifying these patients. DESIGN–Prospective cohort study. SETTING–Eleven hospital outpatient lipid clinics in the United Kingdom. PATIENTS–282 men and 244 women aged 20-74 with heterozygous familial hypercholesterolaemia. MAIN OUTCOME MEASURE–Standardised mortality ratio, all adults in England and Wales being taken as standard (standardised mortality ratio = 100 for standard population). RESULTS–The cohort was followed up for 2234 person years during 1980-9. Fifteen of the 24 deaths were due to coronary heart disease, giving a standardised mortality ratio of 386 (95% confidence interval 210 to 639). The excess mortality from this cause was highest at age 20-39 (standardised mortality ratio 9686; 3670 to 21,800) and decreased significantly with age. The standardised mortality ratio for all causes was 183 (117 to 273) and also was highest at age 20-39 (standardised mortality ratio 902; 329 to 1950). There was no significant difference between men and women. Criteria for measurement of cholesterol concentration in cardiovascular screening programmes (family history, presence of myocardial infarction, angina, stroke, corneal arcus, xanthelasma, obesity, hypertension, diabetes, or any of these) were present in 78% of patients. CONCLUSIONS–Familial hypercholesterolaemia is associated with a substantial excess mortality from coronary heart disease in young adults but may not be associated with a substantial excess mortality in older patients. Criteria for selective measurement of cholesterol concentration in cardiovascular screening programmes identify about three quarters of patients with the clinically overt condition.
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